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Fungal pathogens,Candida albicans,Candida auris
medical mycology, fungal infections, host-pathogen interactions, innate immune responses, metabolic control of infections.
gene regulation, live cell imaging, candidaemia infection model in mice, fungal molecular genetics and cell biology
Systemic model of Candida infection
Candida albicans
Saccharomyces cerevisiae
0000-0001-6252-3104
Ana Traven
Ms. Tricia Lo,Dr. Harshini Weerasinghe,Dr. Manasa Bharthwaj,Dr. Francios Olivier,Ms Irma Tedja,Ms Antara Ghoshal,Ms Orawan Tulyaprawat,Dr. Helen Stoelting
- Candida auris uses nutrient sensing to modulate virulence and host immune responses.
- Infection-induced glucose starvation triggers NINJ1-dependent macrophage lysis and Candida escape.
- Regulation of airway fumarate by host and pathogen promotes Staphylococcus aureus pneumonia.
- Gladiolin produced by pathogenic Burkholderia synergizes with amphotericin B through membrane lipid rearrangements.
- Metabolic homeostasis in fungal infections from the perspective of pathogens, immune cells, and whole-body systems.
- The C-terminal protein interaction domain of the chromatin reader Yaf9 is critical for pathogenesis of Candida albicans.
- The short-chain fatty acid crotonate reduces invasive growth and immune escape of Candida albicans by regulating hyphal gene expression.
- The proteasome regulator Rpn4 controls antifungal drug tolerance by coupling protein homeostasis with metabolic responses to drug stress.
- Disruption of Iron Homeostasis and Mitochondrial Metabolism Are Promising Targets to Inhibit Candida auris.
- The RSC (Remodels the Structure of Chromatin) complex of Candida albicans shows compositional divergence with distinct roles in regulating pathogenic traits.
- Natural Variation in Clinical Isolates of Candida albicans Modulates Neutrophil Responses.
- Metabolic competition between host and pathogen dictates inflammasome responses to fungal infection.
- Mdivi-1 and mitochondrial fission: recent insights from fungal pathogens.
- Central metabolic interactions of immune cells and microbes: prospects for defeating infections.
